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Two of the occupants of this grave were full siblings: a brother (aged 7-9 years old) and a sister (aged 9-11 years old) from whom plague DNA was identified. Not enough of the remains of the third individual were preserved to be able to be able to analyse them as well.
Photo: Vladimiri Bazaliiskii.
For creationists who insist that biological complexity requires an intelligent designer, plague presents an awkward question: what, exactly, was the designer trying to achieve? A parasitic bacterium capable of causing fatal disease in children is certainly biologically impressive. Whether it is evidence of benevolent intelligence is another matter entirely, but it appears to conform to creationists 'proof' of intelligent design - irreducible complexity and complex specified genetic information, both of which ID proponents insist can only be created by an intelligent designer.
Research published in Nature reveals that early strains of Yersinia pestis, the plague bacterium, were already causing deadly outbreaks among hunter-gatherers around Lake Baikal in Siberia approximately 5,500 years ago. These were small, mobile communities: crowded agricultural settlements were evidently not a prerequisite for devastating plague outbreaks.
Researchers detected plague DNA in 18 of 46 individuals examined from four cemeteries. Combined with radiocarbon dating, burial evidence and reconstructed family relationships, the findings point to outbreaks that killed members of the same families, with a particularly striking impact on children. That detection rate is a proportion of the sampled dead, of course, not a measurement of the mortality rate in the living population.
The evolutionary details are especially revealing. These early strains lacked some of the adaptations associated with efficient flea-borne transmission in later plague. Nevertheless, they could cause lethal disease. They also possessed a gene locus encoding a superantigen — a substance capable of provoking excessive immune activation — which is absent from later plague strains. Its contribution to their lethality remains a hypothesis, but the changing genetic repertoire exposes a pathogen with an evolutionary history, rather than an organism equipped from the outset with one fixed set of features.
For intelligent design advocates, this creates a familiar difficulty. If useful biological machinery demonstrates a designer’s foresight, why should machinery that helps pathogens exploit their hosts be exempt from the same argument? Invoking a “Fall” supplies a theological explanation, but it does not establish a biological mechanism or account for the particular genetic changes revealed by ancient DNA.






































